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Tendon and ligament injuries, which typically suffer from slow healing due to limited blood supply, may particularly benefit from this peptides ability to enhance fibroblast activity and collateral blood vessel formation
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Summary Keywords GHK, TGF-1, pulmonary fibrosis, epithelial-to-mesenchymal transition, Smad, collagen Citation Zhou X-M, Wang G-L, Wang X-B, Liu L, Zhang Q, Yin Y, Wang Q-Y, Kang J and Hou G (2017) GHK Peptide Inhibits Bleomycin-Induced Pulmonary Fibrosis in Mice by Suppressing TGF1/Smad-Mediated Epithelial-to-Mesenchymal Transition
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